Task failure during exercise to exhaustion in normoxia and hypoxia is due to reduced muscle activation caused by central mechanisms while muscle metaboreflex does not limit performance
In: Frontiers in Physiology, Jg. 6 (2016)
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Zugriff:
To determine whether task failure during incremental exercise to exhaustion (IE) is principally due to reduced neural drive and increased metaboreflex activation eleven men (22±2 years) performed a 10s control isokinetic sprint (IS; 80 rpm) after a short warm-up. This was immediately followed by an IE in normoxia (Nx, PIO2:143 mmHg) and hypoxia (Hyp, PIO2:73 mmHg) in random order, separated by a 120 min resting period. At exhaustion, the circulation of both legs was occluded instantaneously (300 mmHg) during 10 or 60s to impede recovery and increase metaboreflex activation. This was immediately followed by an IS with open circulation. Electromyographic recordings were obtained from the vastus medialis and lateralis. Muscle biopsies and blood gases were obtained in separate experiments. During the last 10s of the IE, pulmonary ventilation, VO2, power output and muscle activation were lower in hypoxia than in normoxia, while pedaling rate was similar. Compared to the control sprint, performance (IS-Wpeak) was reduced to a greater extent after the IE-Nx (11% lower P
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Task failure during exercise to exhaustion in normoxia and hypoxia is due to reduced muscle activation caused by central mechanisms while muscle metaboreflex does not limit performance
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Autor/in / Beteiligte Person: | eTorres-Peralta, Rafael ; eMorales-Alamo, David ; eGonzalez-Izal, Miriam ; eLosa-Reyna, José ; ePérez-Suárez, Ismael ; eIzquierdo, Mikel ; Jose A Lopez Calbet |
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Zeitschrift: | Frontiers in Physiology, Jg. 6 (2016) |
Veröffentlichung: | Frontiers Media S.A., 2016 |
Medientyp: | academicJournal |
ISSN: | 1664-042X (print) |
DOI: | 10.3389/fphys.2015.00414 |
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